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Cardiologist Alabaster AL

Local resource for Cardiologist in Alabaster. Find addresses and phone numbers of business and services that provide access to Cardiologist in Alabaster, AL.

John David Mc Brayer, MD
(205) 663-5775
1022 1st St N Ste 500
Alabaster, AL
Specialties
Cardiology, Critical Care Medicine-Internal Medicine
Gender
Male
Education
Medical School: Univ Of Ms Sch Of Med, Jackson Ms 39216
Graduation Year: 1978
Hospital
Hospital: Baptist Shelby Med Ctr, Alabaster, Al
Group Practice: Cardiovascular Associates

Data Provided By:
John David McBrayer
(205) 663-5775
1022 1st St N
Alabaster, AL
Specialty
Cardiology, Cardiovascular Disease

Data Provided By:
Dr.John McBrayer
(205) 663-5775
1022 1st St N #500
Alabaster, AL
Gender
M
Speciality
Cardiologist
General Information
Hospital: Shelby & Brookwood
Accepting New Patients: Yes
RateMD Rating
5.0, out of 5 based on 3, reviews.

Data Provided By:
Clyde Dale Elliott
(205) 663-5775
1022 1st St N
Alabaster, AL
Specialty
Cardiology, Cardiovascular Disease

Data Provided By:
Mark Lee Mullens, MD
(205) 663-5775
1004 1st St N Ste 200
Alabaster, AL
Specialties
Cardiology, Internal Medicine
Gender
Male
Education
Medical School: Univ Of Al Sch Of Med, Birmingham Al 35294
Graduation Year: 1989

Data Provided By:
Joyce Rene Koppang, MD
(205) 663-5775
1022 1st St N Ste 500
Alabaster, AL
Specialties
Cardiology
Gender
Female
Education
Medical School: Univ Of Fl Coll Of Med, Gainesville Fl 32610
Graduation Year: 1987
Hospital
Hospital: University Of Alabama Hosp, Birmingham, Al
Group Practice: Birmingham Cardiovascular Grp

Data Provided By:
Clyde Dale Elliott, MD
(205) 663-5775
1022 1st St N Ste 500
Alabaster, AL
Specialties
Cardiology, Internal Medicine
Gender
Male
Education
Medical School: Va Commonwealth Univ, Med Coll Of Va Sch Of Med, Richmond Va 23298
Graduation Year: 1974

Data Provided By:
Thomas Otis Paul Jr, MD
(205) 620-4867
606 1st St N
Alabaster, AL
Specialties
Cardiology
Gender
Male
Education
Medical School: Univ Of Al Sch Of Med, Birmingham Al 35294
Graduation Year: 1978

Data Provided By:
Mark Lee Mullens
(205) 663-5775
1022 1st St N
Alabaster, AL
Specialty
Cardiology, Internal Medicine, Cardiovascular Disease

Data Provided By:
Thomas O Paul
(205) 620-4867
606 1st St N
Alabaster, AL
Specialty
Cardiology

Data Provided By:
Data Provided By:

Restrictive Cardiomyopathy

In restrictive cardiomyopathy, the heart resists filling with blood. Hence the amount of blood pumped out by the heart is not sufficient to meet the body's increased need for the energy and nutrients when the person is exercising. But the heart will be able to provide adequate blood when the person is resting. 

Restrictive cardiomyopathy is the least common form of cardiomyopathy. It has several similarities  with hypertrophic cardiomyopathy.

Causes of Restrictive Cardiomyopathy

Cause is usually not known.

1. The heart muscle is gradually replaced by scar tissue.

2. The heart muscle is infiltrated by abnormal material, such as white blood cells.

3. Amyloidosis

4. Sarcoidosis

5. Hemochromatosis- When the body contains excessive amount of iron, it may accumulate in the heart muscle, as in iron overload.

6. A tumor invading the heart tissue.

Symptoms of Restrictive Cardiomyopathy

  • Shortness of breath

  • Tissue swelling (edema)

  • Abnormal heart rhythms

  • Palpitations

  • Heat failure

  • Chest pain and fainting - These are less likely than in hypertrophic cardiomyopathy

    Diagnosis

    Restrictive cardiomyopathy is often one of the suspected causes of heart failure. 
    The diagnosis of restrictive cardiomyopathy is based largely on a physical examination, an electrocardiogram (ECG), and an echocardiogram.

    Magnetic resonance imaging (MRI) is sometimes used to provide information about the structure of the heart.

    Catheterization of the heart is required to arrive at a precise diagnosis.

    Prognosis

    About 70 percent of people with restrictive cardiomyopathy die within 5 years of when symptoms begin.

    Treatment

    No satisfactory therapy is available.

    Diuretics may actually worsen the condition instead of improving it.

    Drugs normally used in heart failure to reduce the heart's workload may actually reduce the blood pre...

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